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Equally Potent Inhibition of c-Src and Abl by Compounds that Recognize Inactive Kinase ConformationsSEELIGER, Markus A; RANJITKAR, Pratistha; KASAP, Corynn et al.Cancer research (Chicago, Ill.). 2009, Vol 69, Num 6, pp 2384-2392, issn 0008-5472, 9 p.Article

In bcr-abl-positive myeloid cells resistant to conventional chemotherapeutic agents, expression of Par-4 increases sensitivity to imatinib (STI571) and histone deacetylase-inhibitorsBRIEGER, Angela; BOEHRER, Simone; KAI UWE CHOW et al.Biochemical pharmacology. 2004, Vol 68, Num 1, pp 85-93, issn 0006-2952, 9 p.Article

Gène Abl (Abelson) = Abl (Abelson) geneBulletin du cancer. 1999, Vol 86, Num 4, pp 329-330, issn 0007-4551Article

Restriction fragment length polymorphism caused by a deletion within the human c-abl gene (ABL)DE QI XU; GALIBERT, F.Proceedings of the National Academy of Sciences of the United States of America. 1986, Vol 83, Num 10, pp 3447-3450, issn 0027-8424Article

Structural alterations in the carboxyl-terminal domain of the BCRABL gene product activate its fibroblastic transforming potentialSHORE, S. K; LA CAVA, M; SUNEETHA YENDAPALLI et al.The Journal of biological chemistry (Print). 1994, Vol 269, Num 7, pp 5413-5419, issn 0021-9258Article

The chronic myelocytic cell line K562 contains a breakpoint in bcr and produces a chimeric bcr/c-abl transcriptGROSVELD, G; VERWOERD, T; VAN AGTHOVEN, T et al.Molecular and cellular biology (Print). 1986, Vol 6, Num 2, pp 607-616, issn 0270-7306Article

Phosphorylation of synthetic peptides containing Tyr-Met-X-Met motifs by nonreceptor tyrosine kinases in vitroGARCIA, P; SHOELSON, S. E; GEORGE, S. T et al.The Journal of biological chemistry (Print). 1993, Vol 268, Num 33, pp 25146-25151, issn 0021-9258Article

Phosphorylation of the catalytic subunit of type-1 protein phosphatase by the v-abl tyrosine kinaseVILLA-MORUZZI, E; DALLA ZONCA, P; CRABB, J. W et al.FEBS letters. 1991, Vol 293, Num 1-2, pp 67-71, issn 0014-5793Article

Alternative splicing of RNAs transcribed from the human abl gene and from the bcr-abl fused geneSHTIVELMAN, E; LIFSHITZ, B; GALE, R. P et al.Cell (Cambridge). 1986, Vol 47, Num 2, pp 277-284, issn 0092-8674Article

Crystal structure of the T315I abl mutant in complex with the aurora kinases inhibitor PHA-739358MODUGNO, Michele; CASALE, Elena; ISACCHI, Antonella et al.Cancer research (Baltimore). 2007, Vol 67, Num 17, pp 7987-7990, issn 0008-5472, 4 p.Article

Detection of ABL kinase domain mutations with denaturing high-performance liquid chromatographyDEININGER, M. W. N; MCGREEVEY, L; WILLIS, S et al.Leukemia. 2004, Vol 18, Num 4, pp 864-871, issn 0887-6924, 8 p.Article

Post-transcriptional mechanisms in BCR/ABL leukemogenesis: role of shuttling RNA-binding proteinsPERROTTI, Danilo; CALABRETTA, Bruno.Oncogene (Basingstoke). 2002, Vol 21, Num 56, pp 8577-8583, issn 0950-9232, 7 p.Article

Mutagenic analysis of the roles of SH2 and SH3 domains in regulation of the Abl tyrosine kinaseMAYER, B. J; BALTIMORE, D.Molecular and cellular biology (Print). 1994, Vol 14, Num 5, pp 2883-2894, issn 0270-7306Article

Role of the BCR-ABL oncogene in human leukemia : fifteenth Richard and Hinda Rosenthal foundation award lectureWITTE, O. N.Cancer research (Baltimore). 1993, Vol 53, Num 3, pp 485-489, issn 0008-5472Article

A novel human gener closely related to the abl proto-oncogeneKRUH, G. D; KING, C. R; KRAUS, M. H et al.Science (Washington, D.C.). 1986, Vol 234, Num 4783, pp 1545-1548, issn 0036-8075Article

The human cellular abl gene product in the chronic myelogenous leukemia cell line K562 has an associated tyrosine protein kinase activityKLOETZER, W; KURZROCK, R; SMITH, L et al.Virology (New York, NY). 1985, Vol 140, Num 2, pp 230-238, issn 0042-6822Article

Physical and functional interaction between hMSH5 and c-AblWEI YI; LEE, Tai-Hsien; TOMPKINS, Joshua D et al.Cancer research (Baltimore). 2006, Vol 66, Num 1, pp 151-158, issn 0008-5472, 8 p.Article

A limited set of SH2 domains binds BCR through a high affinity phophotyrosine-independent interactionMULLER, A. J; PENDERGAST, A.-M; HAVLIK, . H et al.Molecular and cellular biology (Print). 1992, Vol 12, Num 11, pp 5087-5093, issn 0270-7306Article

Expression of the Transcriptional Repressor Gfi-1 Is Regulated by C/EBPα and Is Involved in Its Proliferation and Colony Formation-Inhibitory Effects in p210BCR/ABL-Expressing CellsLIDONNICI, Maria Rosa; AUDIA, Alessandra; CALABRETTA, Bruno et al.Cancer research (Chicago, Ill.). 2010, Vol 70, Num 20, pp 7949-7959, issn 0008-5472, 11 p.Article

Localization of the cellular oncogenes ABL, SIS, and FES on human germ-line chromosomesJHANWAR, S. C; NEEL, B. G; HAYWARD, W. S et al.Cytogenetics and cell genetics. 1984, Vol 38, Num 1, pp 73-75, issn 0301-0171Article

The ABL Switch Control Inhibitor DCC-2036 Is Active against the Chronic Myeloid Leukemia Mutant BCR-ABLT3151 and Exhibits a Narrow Resistance ProfileEIDE, Christopher A; ADRIAN, Lauren T; O'HARE, Thomas et al.Cancer research (Chicago, Ill.). 2011, Vol 71, Num 9, pp 3189-3195, issn 0008-5472, 7 p.Article

Transcriptional repression of c-Myb and GATA-2 is involved in the biologic effects of C/EBPα in p210BCR/ABL-expressing cellsSOLIERA, Angela Rachele; LIDONNICI, Maria Rosa; FERRARI-AMOROTTI, Giovanna et al.Blood. 2008, Vol 112, Num 5, pp 1942-1950, issn 0006-4971, 9 p.Article

C-Abl kinase inhibitors overcome CD40-mediated dmg resistance in CLL : implications for therapeutic targeting of chemoresistant niches. CommentaryGORDON, John; HALLAERT, Delfine Y. H; JASPERS, Annelieke et al.Blood. 2008, Vol 112, Num 13, issn 0006-4971, 4787-4788, 5141-5149 [11 p.]Article

Expression of the miR-17-92 polycistron in chronic myeloid leukemia (CML) CD34+ cellsVENTURINI, Letizia; BATTMER, Karin; CASTOLDI, Mirco et al.Blood. 2007, Vol 109, Num 10, pp 4399-4405, issn 0006-4971, 7 p.Article

The structure of dasatinib (BMS-354825) bound to activated ABL kinase domain elucidates its inhibitory activity against imatinib-resistant ABL mutantsTOKARSKI, John S; NEWITT, John A; DIANLIN XIE et al.Cancer research (Baltimore). 2006, Vol 66, Num 11, pp 5790-5797, issn 0008-5472, 8 p.Article

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